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Azarias, et al. “A Specific and Essential Role for NA.K-ATPase a 3 in Neurons Co-expressing a 1 a 3.” J Biol Che. 288, no. 4 (January 25, 2013): 2734-43.

Balint B, Stephen CD, Udani V, Sankhla CS, Barad NH, Lang AE, Bhatia KP. “Paroxysmal Asymmetric Dystonic Arm Posturing-A Less Recognized but Characteristic Manifestation of ATP1A3-related Disease.” Mov Disord Clin Pract. 2019 Apr 4; 6(4):312-315. Abstract: https://www.ncbi.nlm.nih.gov/pubmed/31061839

Bizec CL, Nicole S, Panagiotakaki E, Seta N, Vuillaumier-Barrot S. “No Mutation in the SLC2A3 Gene in Cohorts of GLUT1 Deficiency Syndrome-Like Patients Negative for SLCA21 and in Patients with AHC Negative for ATP1A3.” JIMD Rep. 12 (2014):115-120.

Boelman C, Lagman-Bartolome A, MacGregor D, McCabe J, Logan W, Minassian B. “Identical ATP1A3 Mutation Causes Alternating Hemiplegia of Childhood and Rapid-Onset Dystonia Parkinsonism Phenotypes.” Pediatric Neurology (December 2014): 850-853.

Brashear A, Ozelius L, Sweadner K. “ATP1A3 Mutations: What is the Phenotype?” Neurology (February 11, 2014).

Carecchio M, Zorzi G, Ragona F, Zibordi F, Nardocci N. “ATP1A3-related Disorders: An Update.” Eur J Paediatr Neurol. 2018 Mar; 22(2): 257-263. Abstract: https://www.sciencedirect.com/science/article/pii/S1090379817318949?via%3Dihub